Gastrointestinal (GI): NSAIDs that block COX-1 activity have correlations with GI complications because of the inhibition in the synthesis of gastroprotective agents such as PGE2 along with other prostaglandins. This inhibition results in lowered mucin generation by gastric epithelial cells, fewer bicarbonate secretion, and fewer epithelial cell turnover, among other https://williamc246sts9.myparisblog.com/profile